Donn Byrne Intermediate Comprehension Passages Pdf Download Fixed
Donn Byrne Intermediate Comprehension Passages Pdf Download Fixed
Donn Byrne Intermediate Comprehension Passages Pdf Download
Airway inflammation has been defined as consisting of 3 components namely
- active recruitment of cells from the peripheral blood into the airway lumen
- activation of cells already resident within the airway mucosa
- formation of a barrier that inhibits further ingress of cells from the peripheral blood, but also limits the egress of mucus and other mediators from the lamina propria
In addition to these local processes, interactions with cells in the circulation are likely to modify the phenotype of the resident cells – particularly those of the lamina propria. Extravasation may be a major way in which these circulating elements affect the local inflammatory process.
In the past, allergic rhinitis was considered to be a disorder localized to the nose and nasal passages, but current evidence indicates that it may represent a component of a systemic airway disease involving the entire respiratory tract. There are a number of physiological, functional and immunological relationships between the upper (nose, nasal cavity, paranasal sinuses, Eustachian tube, pharynx and larynx) and lower (trachea, bronchial tubes, bronchioles and lungs) respiratory tracts. For example, both tracts contain a ciliated epithelium consisting of goblet cells that secrete mucous, which serves to filter the incoming air and protect structures within the airways. Furthermore, the submucosa of both the upper and lower airways includes a collection of blood vessels, mucous glands, supporting cells, nerves and inflammatory cells. Evidence has shown that allergen provocation of the upper airways not only leads to a local inflammatory response, but may also lead to inflammatory processes in the lower airways, and this is supported by the fact that rhinitis and asthma frequently coexist.
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